Details of a Researcher - UMEDA Satoshi

Papers - UMEDA Satoshi

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Relationship between the Dark Triad personality and the transition in forming interpersonal impression

Keita Masui, Mari Henmi, Nobuhiko Kijima, Satoshi Umeda

INTERNATIONAL JOURNAL OF PSYCHOLOGY 51 909 - 909 2016.07

ISSN 0020-7594

Association between interoceptive accuracy in resting state and emotional state

Yuto Tanaka, Yuri Terasawa, Satoshi Umeda

INTERNATIONAL JOURNAL OF PSYCHOLOGY 51 555 - 555 2016.07

ISSN 0020-7594

Time course and localization of brain activity during numerical processing in grapheme color synesthesia, an ERP and sLORETA study

Midori Shibata, Sa-Ya Fukuzaki, Arisa Sato, Satoshi Umeda

INTERNATIONAL JOURNAL OF PSYCHOLOGY 51 886 - 886 2016.07

ISSN 0020-7594

Temporal characteristics of thought orientation: an ERP study

Yuichi Ito, Midori Shibata, Yuto Tanaka, Yuri Terasawa, Satoshi Umeda

INTERNATIONAL JOURNAL OF PSYCHOLOGY 51 883 - 884 2016.07

ISSN 0020-7594

Effects of aging on interoceptive accuracy and emotional sensitivity

Yuri Terasawa, Satoshi Umeda

INTERNATIONAL JOURNAL OF PSYCHOLOGY 51 555 - 555 2016.07

ISSN 0020-7594

Fear conditioning induced by interpersonal conflicts in patients with dysthymic disorder

Mitsuhiro Tada, Aki Endo, Mika Konishi, Takaki Maeda, Masuru Mimura, Ai Otani, Takuya Takahashi, Yuri Terasawa, Hiroyuki Uchida, Satoshi Umeda

INTERNATIONAL JOURNAL OF NEUROPSYCHOPHARMACOLOGY 19 124 - 124 2016.06

ISSN 1461-1457

fMRIを用いたうつ病に対する認知行動療法の脳神経活動変化に関する予備的検討

片山奈理子, 中川敦夫, 寺澤悠理, 菊地俊暁, 梅田聡, 田渕肇, 三村將

精神神経学雑誌 ((公社)日本精神神経学会) 112th ( 2016特別号 ) S624 - S624 2016.06

ISSN 0033-2658

Sense of agency in competitive situations: Effects of behavioral consequences and others

Yajima Yurie, Tanaka Yuto, Umeda Satoshi

Proceedings of the Japanese Society for Cognitive Psychology (The Japanese Society for Cognitive Psychology) 2016 142 - 142 2016

ある行動について，それを自分が起こしたと感じる感覚を自己主体感という。本研究では，集団での行動のような，誰が実際の行動の主体か不明である状況における主体判断に焦点を当て，自己主体感が行動の結果や共に作業する相手によってどのような違いが見られるか検討した。面識のない他人あるいは知人と参加者2人で，提示される視覚刺激に対し早押しをさせ，ポジティブあるいはネガティブな結果を提示した後，実際の早押しをしたのが誰であったか判断をさせた。その結果，相手が他人の場合は，感情価にかかわらずフィードバックによる変化が大きいときに，自分が主体であったときの正答率が高くなった。一方，相手が知人の場合は，ネガティブな結果のときの主体判断の正答率が高い傾向にあった。これらの結果から，集団における主体判断には他者への親近性が関与することが示された。また，主体判断の誤答が精神疾患傾向と関係があることも示唆された。

Behavioral evidence of delayed prediction signals during agency attribution in patients with schizophrenia

Koreki Akihiro, Maeda Takaki, Fukushima Hirokata, Umeda Satoshi, Takahata Keisuke, Okimura Tsukasa, Funayama Michitaka, Iwashita Satoru, Mimura Masaru, Kato Motoichiro

Psychiatry Research 230 ( 1 ) 78 - 83 2015.11

ISSN 0165-1781

Self-disturbance, a core feature of schizophrenia, recently has been explained from the standpoint of an abnormal sense of agency (SoA). Previous studies showed that aberrant SoA in schizophrenia arise from imprecise predictions about the sensory consequences of actions. However, the nature of the malfunctioning predictions remains unclear. We examined the temporally "delayed" nature of inadequate predictions. We studied 30 patients with schizophrenia and 30 healthy controls. Our original SoA task evaluates explicit experience of the temporal causal relationship between an intentional action and an effect on a computer screen under the presence of temporal biases. We introduced an adaptation with a "trial-by-trial" method that prolonged or shortened the temporal biases. We hypothesized that delayed prediction signals in schizophrenia could lead to a match in timing between predictions and actual outcomes, resulting in self-agency. The adjustment courses to changing temporal biases were evaluated. Patients with schizophrenia continued to feel self-agency even when the adjusted temporal bias was longer than 1000. ms. This result indicated that patient's prediction would be delayed in each trial. Our study empirically showed behavioral evidence for "delayed" prediction signals in a SoA paradigm for the first time.

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Behavioral evidence of delayed prediction signals during agency attribution in patients with schizophrenia

Koreki, Akihiro, Maeda, Takaki, Fukushima, Hirokata, Umeda, Satoshi, Takahata, Keisuke, Okimura, Tsukasa, Funayama, Michitaka, Iwashita, Satoru, Mimura, Masaru, Kato, Motoichiro

PSYCHIATRY RESEARCH 230 ( 1 ) 78 - 83 2015.11

Research paper (scientific journal), ISSN 0165-1781

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Neural mechanisms of emotion regulation: A neuroimaing approach

Umeda Satoshi, Tsukiura Takashi, Kikuchi Toshiaki, Sekiguchi Atsushi

The Proceedings of the Annual Convention of the Japanese Psychological Association (The Japanese Psychological Association) 79 SS-085 - SS-085 2015.09

The neural mechanism of the retrieval of taste preference evoked by visual stimulus: An ERP study.

Tsuji Koki, Shibata Midori, Sato Arisa, Umeda Satoshi

The Proceedings of the Annual Convention of the Japanese Psychological Association (The Japanese Psychological Association) 79 1AM-082 - 1AM-082 2015.09

The effects of social category on face perception: An ERP study

Masui Keita, Terasawa Yu~ri, Shibata Midori, Sato Arisa, Tsuji Koki, Fukuda Haruna, Umeda Satoshi

The Proceedings of the Annual Convention of the Japanese Psychological Association (The Japanese Psychological Association) 79 2PM-028 - 2PM-028 2015.09

Attenuated sensitivity to the emotions of others by insular lesion

Terasawa, Yuri, Kurosaki, Yoshiko, Ibata, Yukio, Moriguchi, Yoshiya, Umeda, Satoshi

FRONTIERS IN PSYCHOLOGY 6 78 - 83 2015.09

Research paper (scientific journal), ISSN 1664-1078

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うつ病性障害における安静時fMRIを用いた認知行動療法による脳機能の変化

菊地俊暁, 寺澤悠理, 梅田聡, 渡邊衡一郎, 加藤元一郎

日本生物学的精神医学会・日本神経精神薬理学会合同年会プログラム・抄録集 (日本生物学的精神医学会・日本神経精神薬理学会) 37回・45回 198 - 198 2015.09

Fear conditioning induced by interpersonal conflicts in healthy individuals

Tada Mitsuhiro, Uchida Hiroyuki, Maeda Takaki, Konishi Mika, Umeda Satoshi, Terasawa Yuri, Nakajima Shinichiro, Mimura Masaru, Miyazaki Tomoyuki, Takahashi Takuya

PLoS ONE 10 ( 5 ) 2015.05

ISSN 1932-6203

Psychophysiological markers have been focused to investigate the psychopathology of psychiatric disorders and personality subtypes. In order to understand neurobiological mechanisms underlying these conditions, fear-conditioning model has been widely used. However, simple aversive stimuli are too simplistic to understand mechanisms because most patients with psychiatric disorders are affected by social stressors. The objective of this study was to test the feasibility of a newly-designed conditioning experiment using a stimulus to cause interpersonal conflicts and examine associations between personality traits and response to that stimulus. Twenty-nine healthy individuals underwent the fear conditioning and extinction experiments in response to three types of stimuli: a simple aversive sound, disgusting pictures, and pictures of an actors' face with unpleasant verbal messages that were designed to cause interpersonal conflicts. Conditioned response was quantified by the skin conductance response (SCR). Correlations between the SCR changes, and personality traits measured by the Zanarini Rating Scale for Borderline Personality Disorder (ZAN-BPD) and Revised NEO Personality Inventory were explored. The interpersonal conflict stimulus resulted in successful conditioning, which was subsequently extinguished, in a similar manner as the other two stimuli. Moreover, a greater degree of conditioned response to the interpersonal conflict stimulus correlated with a higher ZANBPD total score. Fear conditioning and extinction can be successfully achieved, using interpersonal conflicts as a stimulus. Given that conditioned fear caused by the interpersonal conflicts is likely associated with borderline personality traits, this paradigm could contribute to further understanding of underlying mechanisms of interpersonal fear implicated in borderline personality disorder.

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Neurovisceral phenotypes in the expression of psychiatric symptoms

Eccles Jessica A., Owens Andrew P., Mathias Christopher J., Umeda Satoshi, Critchley Hugo D.

Frontiers in Neuroscience 9 ( JAN ) 2015

ISSN 1662-4548

This review explores the proposal that vulnerability to psychological symptoms, particularly anxiety, originates in constitutional differences in the control of bodily state, exemplified by a set of conditions that include Joint Hypermobility, Postural Tachycardia Syndrome and Vasovagal Syncope. Research is revealing how brainbody mechanisms underlie individual differences in psychophysiological reactivity that can be important for predicting, stratifying and treating individuals with anxiety disorders and related conditions. One common constitutional difference is Joint Hypermobility, in which there is an increased range of joint movement as a result of a variant of collagen. Joint hypermobility is over-represented in people with anxiety, mood and neurodevelopmental disorders. It is also linked to stress-sensitive medical conditions such as irritable bowel syndrome, chronic fatigue syndrome and fibromyalgia. Structural differences in 'emotional' brain regions are reported in hypermobile individuals, and many people with joint hypermobility manifest autonomic abnormalities, typically Postural Tachycardia Syndrome. Enhanced heart rate reactivity during postural change and as recently recognised factors causing vasodilatation (as noted post prandially, post exertion and with heat) is characteristic of Postural Tachycardia Syndrome, and there is a phenomenological overlap with anxiety disorders, which may be partially accounted for by exaggerated neural reactivity within ventromedial prefrontal cortex. People who experience Vasovagal Syncope, a heritable tendency to fainting induced by emotional challenges (and needle/blood phobia), are also more vulnerable to anxiety disorders. Neuroimaging implicates brainstem differences in vulnerability to faints, yet the structural integrity of the caudate nucleus appears important for the control of fainting frequency in relation to parasympathetic tone and anxiety. Together there is clinical and neuroanatomical evidence to show that common constitutional differences affecting autonomic responsivity are linked to psychiatric symptoms, notably anxiety.

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Structural brain abnormalities in postural tachycardia syndrome

Umeda Satoshi, Harrison Neil Andrew, Gray Marcus A., Mathias Christopher J., Critchley Hugo D.

Frontiers in Neuroscience (Frontiers Media S.A.) 9 ( JAN ) 2015

ISSN 1662-4548

Postural tachycardia syndrome (PoTS), a form of dysautonomia, is characterized by orthostatic intolerance, and is frequently accompanied by a range of symptoms including palpitations, lightheadedness, clouding of thought, blurred vision, fatigue, anxiety and depression. Although the estimated prevalence of PoTS is approximately 5-10 times as common as the better-known condition orthostatic hypotension, the neural substrates of the syndrome are poorly characterized. In the present study, we used magnetic resonance imaging (MRI) with voxel-based morphometry (VBM) applying the diffeomorphic anatomical registration through exponentiated lie algebra (DARTEL) procedure to examine variation in regional brain structure associated with PoTS. We recruited eleven patients with established PoTS and twenty-three age-matched normal controls. Group comparison of grey matter volume revealed diminished grey matter volume within the left anterior insula, right middle frontal gyrus and right cingulate gyrus in the PoTS group. We also observed lower white matter volume beneath the precentral gyrus and paracentral lobule, right pre- and post-central gyrus, paracentral lobule and superior frontal gyrus in PoTS patients. Subsequent ROI analyses revealed significant negative correlations between left insula volume and trait anxiety and depression scores. Together, these findings of structural differences, particularly within insular and cingulate components of the salience network, suggest a link between dysregulated physiological reactions arising from compromised central autonomic control (and interoceptive representation) and increased vulnerability to psychiatric symptoms in PoTS patients.

Access to Document (DOI)

Neurovisceral phenotypes in the expression of psychiatric symptoms

Eccles Jessica A., Owens Andrew P., Mathias Christopher J., Umeda Satoshi, Critchley Hugo D.

Frontiers in Neuroscience 9 ( FEB ) 2015

ISSN 1662-4548

This review explores the proposal that vulnerability to psychological symptoms, particularly anxiety, originates in constitutional differences in the control of bodily state, exemplified by a set of conditions that include Joint Hypermobility, Postural Tachycardia Syndrome and Vasovagal Syncope. Research is revealing how brain-body mechanisms underlie individual differences in psychophysiological reactivity that can be important for predicting, stratifying and treating individuals with anxiety disorders and related conditions. One common constitutional difference is Joint Hypermobility, in which there is an increased range of joint movement as a result of a variant of collagen. Joint hypermobility is over-represented in people with anxiety, mood and neurodevelopmental disorders. It is also linked to stress-sensitive medical conditions such as irritable bowel syndrome, chronic fatigue syndrome and fibromyalgia. Structural differences in "emotional" brain regions are reported in hypermobile individuals, and many people with joint hypermobility manifest autonomic abnormalities, typically Postural Tachycardia Syndrome. Enhanced heart rate reactivity during postural change and as recently recognized factors causing vasodilatation (as noted post-prandially, post-exertion and with heat) is characteristic of Postural Tachycardia Syndrome, and there is a phenomenological overlap with anxiety disorders, which may be partially accounted for by exaggerated neural reactivity within ventromedial prefrontal cortex. People who experience Vasovagal Syncope, a heritable tendency to fainting induced by emotional challenges (and needle/blood phobia), are also more vulnerable to anxiety disorders. Neuroimaging implicates brainstem differences in vulnerability to faints, yet the structural integrity of the caudate nucleus appears important for the control of fainting frequency in relation to parasympathetic tone and anxiety. Together there is clinical and neuroanatomical evidence to show that common constitutional differences affecting autonomic responsivity are linked to psychiatric symptoms, notably anxiety.

Access to Document (DOI)

Structural brain abnormalities in postural tachycardia syndrome

Umeda Satoshi, Harrison Neil A., Gray Marcus A., Mathias Christopher J., Critchley Hugo D.

Frontiers in Neuroscience 9 ( MAR ) 2015

ISSN 1662-4548

Postural tachycardia syndrome (PoTS), a form of dysautonomia, is characterized by orthostatic intolerance, and is frequently accompanied by a range of symptoms including palpitations, lightheadedness, clouding of thought, blurred vision, fatigue, anxiety, and depression. Although the estimated prevalence of PoTS is approximately 5-10 times as common as the better-known condition orthostatic hypotension, the neural substrates of the syndrome are poorly characterized. In the present study, we used magnetic resonance imaging (MRI) with voxel-based morphometry (VBM) applying the diffeomorphic anatomical registration through exponentiated lie algebra (DARTEL) procedure to examine variation in regional brain structure associated with PoTS. We recruited 11 patients with established PoTS and 23 age-matched normal controls. Group comparison of gray matter volume revealed diminished gray matter volume within the left anterior insula, right middle frontal gyrus and right cingulate gyrus in the PoTS group. We also observed lower white matter volume beneath the precentral gyrus and paracentral lobule, right pre- and post-central gyrus, paracentral lobule and superior frontal gyrus in PoTS patients. Subsequent ROI analyses revealed significant negative correlations between left insula volume and trait anxiety and depression scores. Together, these findings of structural differences, particularly within insular and cingulate components of the salience network, suggest a link between dysregulated physiological reactions arising from compromised central autonomic control (and interoceptive representation) and increased vulnerability to psychiatric symptoms in PoTS patients.

Access to Document (DOI)

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